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Title
Prader‑Willi Syndrome
ImagePath
System
genomic imprinting disorder
Overview
Loss of paternally expressed genes on chromosome 15q11‑q13.
Pathophysiology
70 % paternal deletion; 25 % maternal uniparental disomy; remainder imprinting defects → ↓ hypothalamic satiety signals & endocrine dysfunction.
Presentation
Neonatal hypotonia, feeding difficulty → hyperphagia & obesity in childhood, short stature, hypogonadism, intellectual disability, almond‑shaped eyes.
Diagnosis
Methylation‑specific PCR or FISH for 15q11‑q13.
Management
Caloric restriction, growth hormone therapy, behavioral support.
Epidemiology
≈ 1 : 15 000 births.
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